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Promoting by apamin of the effects of the PAR-2 agonist SLIGRL-NH2 (SLp-NH2) (a) and the PAR-1 agonist TFLLR-NH2 (TFp-NH2) (b) at subeffective doses on gastrointestinal transit in mice. Apamin at 0.01 μmol kg−1 was administered i.p. to the mouse 6 min before i.p. injection of SLIGRL-NH2 at 1 μmol kg−1 or TFLLR-NH2 at 2.5 μmol kg−1 (5 min before amastatin at 2.5 μmol kg−1). Data represent the mean±s.e.mean from 7–8 (a) or 5–6 (b) mice. n.s., not significant.
