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Effect of gap junction uncouplers and CB1 cannabinoid receptor agonists on ACh-induced vasodilator responses in the hindlimb of wild-type mice. Effects of the gap junction uncoupler 18α-glycyrrhetinic acid (aGA; 30 μmol/liter) and the CB1-receptor agonists Δ9-tetrahydrocannabinol (THC; 3 μmol/liter) and HU210 (10 μmol/liter) in the presence (+) or absence (−) of l-NA (300 μmol/liter). Diclofenac (10 μmol/liter) was continuously present during the experiment. CTL, control. n ≥ 5.
