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A number of factors counter-balance the drive to a fully committed Th-2 response to allergens in the fetus. These include the fetal generation of IFN-γ and a maternal supply of soluble CD14 which, in combination with endotoxin, will lead to stimulation of IL-12 and, therefore, IFN-γ production. Maternal lgG antibody which is actively transported across the placenta may block fetal allergan sensitisation.
