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. 2006 Nov;169(5):1686–1700. doi: 10.2353/ajpath.2006.060345

Figure 3.

Figure 3

Aflagellate S. enteritidis causes a hypervirulent phenotype in model systemic murine infection. A: Inflammatory responses in C3H/HeN mice after exposure to S. enteritidis (WT-2 or aflagellate-2). H&E-stained frozen sections. i: Ileum, 6 days after infection showing normal crypt histology. ii–iv: Ileum, 12 days after infection showing edema (arrows) (ii) and cellular infiltrate between crypts (iii) and (iv) jejunum, 12 days after infec-tion showing the presence of neutrophils (arrows) in the lamina propria (LP) and transepithelial migration of PMNs into crypt abscesses. B: Ileal MPO activity. Aflagellate-2-infected versus control, or WT-2, *P < 0.05. C: Survival (percent) of C3H/HeN mice after oral infection with 108 CFU with WT-2 or aflagellate-2 as indicated. Scale bars = 10 μm (i, ii); 10 μm (iii); 25 μm (iv).