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. 2007 Jan 24;26(2):402–411. doi: 10.1038/sj.emboj.7601519

Figure 5.

Figure 5

B56γ3 inhibits cell proliferation in a p53-dependent manner. Representatives of cell proliferation of the HCT116 human colon cancer cell lines (A) or the H1299 human lung cancer cell line (B) transfected with either B56γ3 or a control CMV empty vector. H1299+p53: H1299 transfected with wild-type p53; H1299+T55D: H1299 transfected with T55D. Error bars show average±s.d. from triplicate plates in one representative experiment. (C) Immunoblotting of the transfected HA-B56γ3, endogenous B56γ3/B56γ2, p53 and p21 proteins in HCT116 and H1299 cells. M: empty vector-transfected cell lysate at seeding. (D) Cell doubling times show average±s.d. from three independent experiments. EV: empty vector.