Table 7.
Genes that serve as markers for acute inflammation.
| Slc26a4+/+ | Slc26a4-/- | |||||||
| No. | Gene | Description | Intensity | P | Intensity | P | Fold | Direction |
| 1 | Il1a | Interleukin 1a | 29 | 0/3 | 44 | 0/3 | ||
| 2 | Il1b | Interleukin 1b | 173 | 0/3 | 156 | 0/3 | ||
| 3 | Il6 | Interleukin 6 | 14 | 0/3 | 31 | 0/3 | ||
| 4 | Il12a | Interleukin 12a | 126 | 0/3 | 96 | 0/3 | ||
| 5 | Il12b | Interleukin 12b | 70 | 1/6 | 73 | 1/6 | ||
| 6 | Il10 | Interleukin 10 | 116 | 3/3 | 87 | 3/3 | ||
| 7 | Ifng | Interferon, gamma | 71 | 1/3 | 24 | 0/3 | ||
| 8 | Tnf | Tumor necrosis factor, alpha | 54 | 0/3 | 50 | 0/3 | ||
| 9 | Nos2 | Nitric oxide synthase, inducible | 19 | 0/3 | 27 | 0/3 | ||
Genes that can serve as markers for acute inflammation (1–9) are listed. For parameters 'P', 'Fold' and 'Direction', see the Gene array section in Methods. Fold changes are given when a gene was called Present (P) in Slc26a4+/- or Slc26a4-/- stria vascularis in at least half of the available probes. The direction of the fold change is only given when it exceeded 1.30. Fold changes lower than 1.30 were not considered significant.