TABLE 1.
Genotype | n | Frequency of abnormal innervation (%) |
---|---|---|
UAS-comm [2PY→AY]/24B-GAL4 | 83 | 42.2b |
UAS-comm [10K→R]/24B-GAL4 | 88 | 35.2b |
UAS-comm+/24B-GAL4 (control 1) | 65 | 20.0c |
+/24B-GAL4 (control 2) | 76 | 3.9 |
UAS-comm [(2PY→AY)-monoUb]/24B-GAL4 | 97 | 19.6 |
UAS-comm [(10K→R)-monoUb]/24B-GAL4 | 86 | 23.3 |
commLOF/+ | 108 | 26.9 |
UAS-comm10K→R/5-GAL4; commΔe39/+ | 72 | 47.2 |
The following P values were determined by Fisher's exact test (two-tailed): 2PY→AY versus WT Comm (control 1), P < = 0.0048; 10K→R versus WT Comm (control 1), P < = 0.0475; 2PY→AY versus +/24B-GAL4 (control 2), P < = 0.0001; 10K→R versus +/24B-GAL4 (control 2), P < = 0.0001; WT Comm (control 1) versus +/24B-GAL4 (control 2), P < = 0.0032; 10K→R-monoUb versus 24B-GAL4 (control 2), P < = 0.0005; 2PY→AY-monoUb versus 24B-GAL4 (control 2), P < = 0.0041; 10K→R-monoUb versus Comm WT (control 1), P < = 0.84; 2PY→AY-monoUb versus Comm WT (control 1), P < = 0.6938; 10K→R in comm heterozygote background versus comm heterozygote, P < = 0.0066; 10K→R in comm heterozygote background versus 10K→R in WT background, P < = 0.1464.
Significance was at the P = 0.05 level with respect to the +/24B-GAL4 and UAS-comm+/24B-GAL4 controls.
Significance was at the P = 0.05 level with respect to the +/24B-GAL4 control.