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. 2005 Jun;140(3):427–435. doi: 10.1111/j.1365-2249.2005.02785.x

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© 2005 British Society for Immunology

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Fig. 5 — Metabolic activity of lymphocytes from SJL (a) and C57BL/6 (b) mice induced by untreated (□) or antileptin antibody-treated () adipocyte supernatants previously incubated with conditioned media (CM) from lipopolysaccharide (LPS)-, peptidoglycan (PEP)- and lactobacillus extracts-treated macrophages. Lymphocytes were incubated with antileptin-treated supernatants for 24 h and metabolic activity was measured by the mitochondrial dehydrogenase activity (MTS/PMS) test. Experiments were conducted in triplicate. These results are representative of two similar experiments. *P ≤ 0·05; **P ≤ 0·01; ***P ≤ 0·001.

Inline graphic — Metabolic activity of lymphocytes from SJL (a) and C57BL/6 (b) mice induced by untreated (□) or antileptin antibody-treated () adipocyte supernatants previously incubated with conditioned media (CM) from lipopolysaccharide (LPS)-, peptidoglycan (PEP)- and lactobacillus extracts-treated macrophages. Lymphocytes were incubated with antileptin-treated supernatants for 24 h and metabolic activity was measured by the mitochondrial dehydrogenase activity (MTS/PMS) test. Experiments were conducted in triplicate. These results are representative of two similar experiments. *P ≤ 0·05; **P ≤ 0·01; ***P ≤ 0·001.