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. 2006 Mar;143(3):550–559. doi: 10.1111/j.1365-2249.2006.03010.x

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© 2006 British Society for Immunology

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Fig. 1 — Stimulation of monocyte-derived dendritic cells (DC) with heat shock protein (HSP)70, CD40L or lipopolysaccharide (LPS) producing (a) tumour necrosis factor (TNF)-α and (b) interleukin (IL)-12 in Crohn's disease (CD) □ (n = 16 and 12), ulcerative colitis (UC) (n = 9 and 8) and healthy controls ▪ (n = 13 and 14), respectively; statistical analyses were performed between CD and controls and UC and controls for each stimulant (*P < 0·05, **P < 0·02). Stimulation of monocytes in these groups producing (c) TNF-α (n = 10–16) and (d) IL-12 (n = 7–9), respectively.

Inline graphic — Stimulation of monocyte-derived dendritic cells (DC) with heat shock protein (HSP)70, CD40L or lipopolysaccharide (LPS) producing (a) tumour necrosis factor (TNF)-α and (b) interleukin (IL)-12 in Crohn's disease (CD) □ (n = 16 and 12), ulcerative colitis (UC) (n = 9 and 8) and healthy controls ▪ (n = 13 and 14), respectively; statistical analyses were performed between CD and controls and UC and controls for each stimulant (*P < 0·05, **P < 0·02). Stimulation of monocytes in these groups producing (c) TNF-α (n = 10–16) and (d) IL-12 (n = 7–9), respectively.