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. Author manuscript; available in PMC: 2007 Mar 6.
Published in final edited form as: J Steroid Biochem Mol Biol. 2006 Nov 7;102(1-5):128–138. doi: 10.1016/j.jsbmb.2006.09.003

Fig. 3.

Fig. 3

Growth inhibition of MCF-7/RAL2 tumors in response to E2 and FUL. Twenty ovariectomized athymic nude mice were bilaterally injected in the axillary mammary fat pads with 107 MCF-7/RAL2 cells grown in culture and separated into 4 treatment groups of 5 mice each (10 tumors per group) corresponding to 1.5 mg/day RAL po, 0.3 cm E2 capsule sc, 2 mg/day FUL sc, and Control (no treatment). The data shown represent the average tumor cross-sectional area (cm2) per group ± SE. The cross-sectional area of RAL-treated MCF-7/RAL2 tumors was significantly different from E2-treated, FUL-treated and Control-treated tumors (all P-values = < 0.0001).