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. 2007 Apr;175(4):1665–1679. doi: 10.1534/genetics.106.067918

TABLE 3.

Timing of anaphase onset in the suppressor mutants

NEBD–anaphase Anaphase–NER NEBD–NER
Embryo s ± SE (n) s ± SE (n) s ± SE (n)
Wild type 154.5 ± 4.3 (6) 143.2 ± 8.2 (6) 297.7 ± 5.1 (6)
F2unc-46(e177) mdf-1(gk2) 177.2 ± 14.2 (5) 127.6 ± 6.1 (5) 304.8 ± 12.9 (6)
unc-46(e177) mdf-1(gk2); h1960 464.2 ± 13.6 (5)* 124.2 ± 2.6 (5) 588.4 ± 14.2 (5)*
unc-46(e177) mdf-1(gk2); h1983 352.2 ± 5.9 (5)* 158.2 ± 3.3 (5) 510.4 ± 5.3 (5)*
unc-46(e177) mdf-1(gk2) h1992 252.8 ± 11.2 (5)* 144.6 ± 11.4 (5) 397.4 ± 7.5 (5)*
unc-46(e177) mdf-1(gk2); h1988ts 344.0 ± 14.1 (5)* 137.8 ± 8.1 (5) 481.8 ± 7.2 (5)*
unc-46(e177) mdf-1(gk2); h1987 178.0 ± 8.1 (5) 124.6 ± 3.5 (5) 302.6 ± 8.5 (5)
unc-46(e177) mdf-1(gk2); h1958 177.5 ± 11.2 (6) 138.7 ± 4.8 (6) 316.2 ± 9.5 (6)
unc-46(e177) mdf-1(gk2) h1985 158.8 ± 8.1 (5) 149.2 ± 6.6 (5) 308.0 ± 8.3 (5)

Suppressor strains that carry ruIs32, an integrated H2B∷GFP transgene (Praitis et al. 2001), were constructed and analyzed for anaphase onset. Asterisks denote a significant difference as shown by P-values of Student's t-test statistic.