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. 2006 Jun;55(6):782–787. doi: 10.1136/gut.2005.080523

Table 2 Review of the literature concerning IgH clonality in chronic gastritis.

Reference Entity Cases with Ig clonality
Isaacson (pers commun) Chronic gastritis 0–1% (n = 300)
Sukpanichnant6 Chronic gastritis 0% (n = 7)
Savio18 Hp gastritis with reactive lymphoid infiltrate 4% (n = 55)
De Mascarel19 Hp gastritis with duodenal ulcer 1% (n = 320)
Saxena9 Hp gastritis with lymphoid hyperplasia 50% (n = 20)
Hsi20 Chronic gastritis 13% (n = 41)
Sorrentino10 Hp positive gastritis 35% (n = 20)
Hp negative gastritis 40% (n = 5)
Torlakovic7 Hp gastritis with lymphoid hyperplasia 85% (n = 20)
Hp gastritis without lymphoid hyperplasia 10% (n = 20)
El‐Zimaity11 Chronic gastritis 38% (n = 39)
Miyamoto21 Follicular gastritis 43% (n = 60)
Non‐follicular gastritis 17% (n = 60)
Hiyama22 Hp chronic gastritis 6% (n = 116)
Subgroup with marked atrophy 18% (n = 40)
Subgroup with marked intestinal metaplasia 20% (n = 20)
Rudolph12 Hp gastritis 20% (n = 15)
Ferraccioli23 Patients with Sjögren syndrome 33% (n = 21)
Various forms of gastritis 22% (n = 80)
Wuendisch13 Wotherspoon 0 0% (n = 24)
Wotherspoon 1 4% (n = 24)
Wotherspoon 2 24% (n = 21)
Wotherspoon 3 29% (n = 14)
Wotherspoon 4 35% (n = 23)

Ig, immunoglobulin; Hp, Helicobacter pylori.