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. Author manuscript; available in PMC: 2008 Mar 1.
Published in final edited form as: Neuropharmacology. 2007 Jan 29;52(4):1114–1123. doi: 10.1016/j.neuropharm.2006.11.011

Figure 2.

Figure 2

On postnatal day 90, ApoER2 and VLDLR mutant mice were tested for deficits in acoustic (Pp8, 73 dB) and multimodal (Ppuff) trials of prepulse inhibition. The latter task consisted of a combination of acoustic prepulses with a tactile (airpuff) startle stimulus. Values represent group means (± SEM). Startle magnitudes (arbitrary units) are inset in graph. * indicates significantly different compared to wildtype and heterozygous mice, p < 0.05. WT = Wildtype (ApoER2, n = 22; VLDLR, n = 22); HT = Heterozygote (ApoER2, n = 31; VLDLR, n = 52); KO = Homozygous knockout (ApoER2, n = 26; VLDLR, n = 39).