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. 2007 May 2;80(6):1115–1124. doi: 10.1086/518445

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© 2007 by The American Society of Human Genetics. All rights reserved.

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Figure 5. — A, Association between carriage of the ATAG haplotype and in vivo lipolysis measured as plasma glycerol corrected for body fat. The blackened bar represents the 124 subjects carrying ATAG; the unblackened bar represents the 1,771 subjects not carrying ATAG. B, Association between carriage of the ATAG haplotype and in vivo lipolysis measured as plasma free fatty acids corrected for body fat. The blackened bar represents the 100 subjects carrying ATAG; the unblackened bar represents the 1,494 subjects not carrying ATAG. C, Association between carriage of the ATAG haplotype and adipocyte lipolytic sensitivity (pD2) of different adrenergic agonists. The blackened bar represents the 14 subjects carrying ATAG; the unblackened bar represents the 136 subjects not carrying ATAG. Analyzed subjects came from samples 1 and 2. Groups were compared by ANCOVA, with age as covariate in panel A and with age and BMI as covariates in panel B.