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. 2007 May;148(2):230–240. doi: 10.1111/j.1365-2249.2007.03336.x

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Fig. 4 — (a,b) Monitoring of immunoglobulin A (IgA) tissue transglutaminase (tTG) autoantibody levels and avidities in children genetically predisposed to coeliac disease (C). Serum samples from 10 children diagnosed with CD (a) were analysed for autoantibody level (•) and avidity (○) during a period extending from seroconversion to 3·5–8·1 months after duodenal biopsy (x), diagnosis of CD and starting gluten-free diet (arrow). In two children (no. 13723 and no. 15805), gluten-free diet was started after the second biopsy. Similarly, autoantibody titres and avidities of follow-up serum samples from 10 children with transient or fluctuating IgA–anti-tissue transglutaminase (TGA) values (b) were measured during the periods of seropositivity. For children with fluctuating autoantibodies, negative titres are also shown. The dotted lines indicate the cut-off value of the fluorometric IgA–TGA assay.

Fig. 4 — (a,b) Monitoring of immunoglobulin A (IgA) tissue transglutaminase (tTG) autoantibody levels and avidities in children genetically predisposed to coeliac disease (C). Serum samples from 10 children diagnosed with CD (a) were analysed for autoantibody level (•) and avidity (○) during a period extending from seroconversion to 3·5–8·1 months after duodenal biopsy (x), diagnosis of CD and starting gluten-free diet (arrow). In two children (no. 13723 and no. 15805), gluten-free diet was started after the second biopsy. Similarly, autoantibody titres and avidities of follow-up serum samples from 10 children with transient or fluctuating IgA–anti-tissue transglutaminase (TGA) values (b) were measured during the periods of seropositivity. For children with fluctuating autoantibodies, negative titres are also shown. The dotted lines indicate the cut-off value of the fluorometric IgA–TGA assay.