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A model depicting the interplay between miRNAs and the t-trkC isoform in RA-induced neuronal differentiation. In undifferentiated SK-N-BE cells, the t-trkC protein level is 2-fold higher than that of the full-length isoform (Left). Upon RA treatment the overexpression of miRNAs causes a reduction of the truncated form, which correlates with a decrease in cell proliferation (Right).
