Table 3.
Treatment, time (h) | Density of pyramidal cells in the hippocampal CA3 subfield, Nv, ipsilateral; mean × 106/mm3 ± SEM | Density of pyramidal cells in the hippocampal CA3 subfield, Nv, contralateral; mean × 106/mm3 ± SEM | Neurons, mean ± SEM | % | n |
---|---|---|---|---|---|
Sham | 0.1899 ± 0.0019 | 0.1891 ± 0.0020 | 123,435 ± 1,235 | 100 | 14 |
Vehicle | 0.1343 ± 0.0024 | 0.1899 ± 0.0018 | 85,852 ± 1,534 | 70 | 14 |
CPP, −2, −1, 0 | 0.1579 ± 0.0059 | 0.1887 ± 0.0016 | 102,635 ± 3,835219 | 83 | 8 |
CPP, 1, 2, 3 | 0.1088 ± 0.0144 | 0.1883 ± 0.0023 | 66,368 ± 8,784*** | 54 | 8 |
CPP, 4, 5, 6 | 0.1162 ± 0.0051 | 0.1879 ± 0.0018 | 69,720 ± 3,061*** | 56 | 9 |
CPP, 7, 8, 9 | 0.1233 ± 0.0033 | 0.1839 ± 0.0016 | 73,981 ± 1,980** | 60 | 10 |
CPP, 10, 11, 12 | 0.1313 ± 0.0068 | 0.1876 ± 0.0013 | 84,032 ± 4,352 | 68 | 6 |
NBQX, −2, −1, 0 | 0.1871 ± 0.0043 | 0.1875 ± 0.0017 | 115,992 ± 2,666219 | 94 | 8 |
NBQX, 1, 2, 3 | 0.1694 ± 0.0035 | 0.1883 ± 0.0019 | 106,720 ± 2,205219 | 86 | 8 |
NBQX, 4, 5, 6 | 0.1624 ± 0.0018 | 0.1891 ± 0.0025 | 100,676 ± 1,116219 | 82 | 8 |
NBQX, 7, 8, 9 | 0.1561 ± 0.0016 | 0.1877 ± 0.0019 | 96,774 ± 1,001219 | 78 | 7 |
NBQX, 10, 11, 12 | 0.1412 ± 0.0021 | 0.1882 ± 0.0018 | 86,153 ± 1,281 | 70 | 8 |
Sham + CPP | 0.1885 ± 0.0027 | 0.1892 ± 0.0013 | 120,650 ± 1,742 | 98 | 8 |
Sham + NBQX | 0.1888 ± 0.0034 | 0.1885 ± 0.0023 | 120,045 ± 2,169 | 97 | 8 |
Treatment with CPP, 3 × 30 mg/kg, and NBQX, 3 × 30 mg/kg, was initiated 2 h before or 1, 4, 7, or 10 h after traumatic cortex injury. To quantitatively assess neuronal loss in the hippocampal CA3 subfield in Fischer 344 rats 3 days after injury, a stereological disector technique was used to estimate Nv. Morphometric analysis revealed that the volume of the hippocampal CA3 subfield in rats subjected to traumatic brain injury and treatment with CPP or NBQX did not significantly differ from that in rats subjected to brain trauma and vehicle and ranged from 0.61 to 0.64 mm3. The differences between vehicle- and drug-treated rats in the numbers of neurons remaining in the hippocampus after injury were analysed statistically by means of Student's t test. **,
, P < 0.01; ***,
, P < 0.001 vs. vehicle-treated rats. Stars indicate significant increase in neurodegeneration, while crosses indicate neuroprotection. Monitoring of blood pressure and arterial blood gases in rats subjected to head trauma and systemic treatment with either CPP (n = 8) or NBQX (n = 12) showed no evidence of cardiorespiratory compromise.