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. 2007 Jun 5;104(24):10086–10091. doi: 10.1073/pnas.0610799104

Table 2.

Fate of P5.p after ablation of P(6,7).p

Strain P5.p 1° (n) Percent P5.p 1° 90% CI, %
A, PS1843 (Wash) 7 (33) 21 10–33
B, RS106 (Pol) 22 (51) 43 32–55
C, Pol × Wash F1 3 (27) 11 1–21
D, Pol × Wash F2 5 (16) 31 12–50
E, RIL 12B 0 (12) 0
F, RIL 14B 0 (6) 0
G, RIL 10A 2 (19) 11 0–22
H, RIL 18C 2 (11) 18 0–37
I, RIL 9A 3 (13) 23 4–42
J, RIL 2A 4 (13) 31 10–52
K, RIL 16C 2 (6) 33 2–65
L, RIL 3A 5 (14) 36 15–57
M, RIL 13B 5 (14) 36 15–57
N, RIL 25B 5 (12) 42 18–65
O, RIL 1A 5 (11) 45 21–70
P, RIL 11B 5 (11) 45 21–70
Q, RIL 17C 5 (10) 50 24–76
R, RIL 4A 4 (8) 50 21–79
S, RIL 20C 7 (11) 64 40–87
T, RIL 19C 13 (14) 93 82–100
Average (16 RILs) 4.2 (11.6) 36
U, RIL C22 12 (13) 92 80–100
V, RIL A8 16 (18) 89 77–100
    W, RIL A8BC7 19 (23) 83 70–96
    X, RIL A8BC10 15 (17) 88 75–100
    Y, RIL A8BC18 11 (12) 92 79–100

P(6,7).p were ablated 0–1 h after hatching, and the cell lineage of P5.p was analyzed as described in ref. 6. A, B, homozygous and isogenic laboratory strains. C, F1 animals. D, segregating F2 population. E–V, homozygous recombinant inbred lines. W−Y, Second-generation RILs. See text for details. Wash, Washington; Pol, Poland. CI, confidence interval.