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. 2003 Dec;163(6):2585–2593. doi: 10.1016/S0002-9440(10)63613-9

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Figure 1. — PrP^res from patients with prion diseases and prion-inoculated Ki-ChM mice A: PrP^res type 1 extracted from MM1 sCJD (H-3, lane 1). PrP^res type 2 from MM2-T sCJD (NK, lane 2), and vCJD (96/02, lane 3). B: PrP^res isolated from Ki-ChM mice inoculated with MM1 sCJD (H-3, lane 2), MM2-T sCJD (NK, lane 4), and vCJD (96/02, lane 6) are shown along with those from the respective inocula (H-3, lane 1; NK, lane 3; 96/02, lane 5). Ki, PrP^res isolated from Ki-ChM mice; In, PrP^res isolated from inocula. C: Deglycosylated PrP^res isolated from Ki-ChM mice inoculated with human prion diseases (the same samples as shown in B): MM1 sCJD, lane 1; MM2-T sCJD, lane 2; vCJD, lane 3.