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. 2003 Dec;163(6):2277–2287. doi: 10.1016/s0002-9440(10)63585-7

Table 2.

β-Catenin Gene Mutations in Skin Tumors Correlate with Matrical Differentiation and TCF-1 and MITF-M Activation

Histologic diagnosis C terminus β-catenin Abs % reactive nuclei αABC pattern β-Catenin mutations TCF-1 % reactive nuclei MITF-M % reactive nuclei
PMX 80 Loss D32N(GAC-AAC) 10 30
PMX 85 Loss G34V(GGA-GTA) 30 60
PMX 85 N+ D32Y(GAC-TAC) 0 40
PMX 85 N+ D32Y(GAC-TAC) 20 30
PMX 90 Loss G34R(GGA-AGA) 20 50
PMX 90 Loss 33 nt insertion after codon 32 5 40
PMX 90 Loss D32E(GAC-GAG)T41A(ACC-GCC) 10 30
PMX 90 Loss D32N(GAC-AAC) 20 40
PMX 90 N+ D32G(GAC-GGC) 20 50
PMX 90 Loss I35S(ATC-AGC) 40 40
PMX 90 N+ D32G(GAC-GGC) 30 40
PMX 90 N+ D32Y(GAC-TAC) 25 50
PMX 90 N+ D32Y(GAC-TAC) 30 20
PMX 90 Loss T41I(ACC-ATC) 30 40
PMX 90 Loss S37F(TCT-TTT) 30 40
PMX 90 N+ S33F(TCT-TTT) 30 50
PMX 90 Loss D32N(GAC-AAC) 20 40
PMX 90 Loss D32N(GAC-AAC) 40 70
TE-MD (TE with matrical differentiation) 40 Loss T41A(ACC-GCC) 10 30
SCC-MD (SCC with matrical differentiation) 80 Loss S37F(TCT-TTT) 10 20

Among 83 skin tumors tested, only 20 harbored β-catenin gene mutations.

These tumors included 18 pilomatricomas and 2 tumors showing peculiar signs of matrical differentiation (1 trichoepithelioma, TE-MD, and 1 squamous cell carcinoma, SCC-MD).