Figure 5.

Dependence of of FN-induced intracellular Ca²⁺ signaling on PTK and G-proteins. Blastocysts were treated with 10 μM genistein, daidzein or suramin and then exposed to FN-120 in the continued presence of the same inhibitor. Embryos treated with vehicle served as controls. Some embryos were not exposed to FN-120 or inhibitor (BSA). Intracellular Ca²⁺ levels (A) and ΔFN-binding activities (B, C) were monitored and reported as in Fig. 2. *, P < 0.05, compared to Control.