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Diesel exhaust particles (DEP) increased the release of soluble ICAM-1 into media. DEP showed a dose-dependent stimulatory effect on sICAM-1 release when evaluated after 24 h, which was significantly suppressed by pretreatment with pyrrolidine dithiocarbamate (PDTC; 10 mm), N-acetyl cysteine (NAC; 10 mm) and SB203580 (10 μm). *P < 0·05 compared with naive cells; **P < 0·05 compared with DEP (50 μg/ml)-treated cells, anova.
