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. Author manuscript; available in PMC: 2008 Jun 1.
Published in final edited form as: Cell Signal. 2007 Jan 9;19(6):1150–1162. doi: 10.1016/j.cellsig.2006.12.009

Fig. 1.

Fig. 1

Exposure to Aβ1-42 leads to progressive neuronal injury and is accompanied by an initial increase in expression of Wnt1. (A) Neuronal survival was progressively decreased with increased concentration of Aβ1-42 (*P<0.01 vs. untreated control). (B) Exposure to Aβ1-42resulted in increased Wnt1 expression at 1 hour (h) and significantly at 6 hours (h) following Aβ1-42application, but loss of Wnt1 by 24 hours (h) with expression levels comparable to untreated control (*P<0.01 vs. control). Total Akt1 expression was not altered by Aβ1-42 exposure over a 24 hour course. Each data point represents the mean and ±SEM from 6 experiments.