Skip to main content
. Author manuscript; available in PMC: 2007 Jul 17.
Published in final edited form as: Neuropsychologia. 2006;44(1):130–139. doi: 10.1016/j.neuropsychologia.2005.03.026

Fig. 1.

Fig. 1

Representative magnetic resonance scans of amnesic participants; all scans shown in the radiologic view (left/right reversal). (A) T1 coronal view through the body of the hippocampus in a representative individual who became amnesic following hypoxic brain injury. There is significant bilateral hippocampal atrophy (arrow points to left hippocampus) and enlargement of the temporal horns of the lateral ventricles. (B) T2 coronal scan of another amnesic individual, whose hypoxic episode resulted in bilateral hippocampal damage (right arrow points to left hippocampus) and extensive enlargement of the temporal horns, cerebral atrophy (left arrow) and ventricular enlargement due to diffuse atrophic changes. (C and D) T2 axial scan of a representative individual who became amnesic following ACoA aneurysm rupture. Right arrow in (C) indicates bilateral damage to basal forebrain and right arrow in (D) shows predominantly left frontal damage. Left arrows in (C) and (D) point to clip artifact.