Skip to main content
. Author manuscript; available in PMC: 2007 Dec 1.
Published in final edited form as: Biochem Biophys Res Commun. 2006 Oct 6;350(4):1063–1068. doi: 10.1016/j.bbrc.2006.09.158

Figure 4. JNK1 deficiency did not alter the protein expression of intracellular signaling proteins involved in skeletal muscle glucose metabolism.

Figure 4.

In mouse gastrocnemius muscle, there was no significant change in the protein expression of c‐jun‐N‐terminal kinase 2 (JNK2), insulin receptor substrate‐1 (IRS‐1), Akt, AS160, AMP‐activated protein kinase (AMPK), acetyl‐CoA‐carboxylase (ACC), glycogen synthase kinase‐3α/β (GSK3α/β), glycogen synthase (GS), glucose transporter 1 (GLUT1) and glucose transporter 4 (GLUT4) between wild‐type and JNK1 knockout (JNK1 KO) mice.