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. 2007 Jul 10;35(14):4869–4881. doi: 10.1093/nar/gkm517

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© 2007 The Author(s)

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/2.0/uk/) which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 2. — Abilities of versions of TFIIIA containing disruptions of two zinc fingers to support TF activity. (A) Abilities of mutant versions of TFIIIA to support in vitro transcription of the 5 S RNA gene. For simplicity, versions of TFIIIA containing histidine-to-asparagine substitutions in multiple fingers are named by the numbers of the disrupted fingers; for example, 3/5 represents TFIIIA with disruptions in zinc fingers 3 and 5 [TFIIIA(H126N/H181N)]. See Figure 1D for details. (B) Abilities of versions of TFIIIA containing disruptions of two zinc fingers to support cell viability. See Figure 1G for details.