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. 2007 Sep 1;21(17):2196–2204. doi: 10.1101/gad.440307

Figure 4.

Figure 4.

Mutations in jkd cause ectopic divisions and misexpression of SCR in the ground tissue. Longitudinal (a,b) and radial (c–f) confocal sections of 3-dpg wild-type root carrying pSCR∷YFP (c) or pSCR∷SCR:GFP (e) with two layers of ground tissue and jkd-i (d,f) roots with ectopic periclinal divisions in cortex cells (arrowhead). Arrow marks QC cells and asterix mark the endodermal cells. (g–j) Longitudinal confocal sections of pSCR∷FPER in 3-dpg wild-type (g), jkd-i (h,i), and jkd-4 roots (j). SCR is expressed in endodermal cells and is absent from cortex cells in wild type. Ectopic periclinal cortex divisions (arrowhead) in jkd mutants coincide with a reduction of SCR promoter activity in adjacent endodermis cells and induction of SCR promoter activity in the middle ground tissue layer. (k) Lateral root primordium emerging from the pericycle in jkd-4. (l–s) Root tissue sections from 10-d-old wild type (l) ([ep] epidermis; [c] cortex; [e] endodermis; [v] vascular bundle); jkd-4 (m); mgpi (n); jkd-4mgpi (o) (note that cell number in the ground tissue layers is restored to wild type); scr-4 (p); jkd-4scr-4 (q); shr-2 (r); and jkd-4shr-2 (s).