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. 2007 Sep 6;4(4):223–231. doi: 10.7150/ijms.4.223

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© Ivyspring International Publisher. This is an open-access article distributed under the terms of the Creative Commons License (http://creativecommons.org/licenses/by-nc-nd/3.0/). Reproduction is permitted for personal, noncommercial use, provided that the article is in whole, unmodified, and properly cited.

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Fig 4 — Induction of estradiol down-regulation in Chlamydia trachomatis infected trophoblast. The cellular estradiol level of infected trophoblast (-♦-) compared with uninfected cells (-■-) is depicted (A). There was significant decline in estradiol production (p < 0.01 * ) in infected trophoblast. The time-course level of aromatase production is represented in B & C. There was a significant increase in expression of the enzyme (p < 0.05 *) in infected trophoblast (-♦-) compared with uninfected trophoblast (-■-). All values represent means ± SEM (n = 3).

Fig 4 — Induction of estradiol down-regulation in Chlamydia trachomatis infected trophoblast. The cellular estradiol level of infected trophoblast (-♦-) compared with uninfected cells (-■-) is depicted (A). There was significant decline in estradiol production (p < 0.01 * ) in infected trophoblast. The time-course level of aromatase production is represented in B & C. There was a significant increase in expression of the enzyme (p < 0.05 *) in infected trophoblast (-♦-) compared with uninfected trophoblast (-■-). All values represent means ± SEM (n = 3).