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. 2007 Sep 6;4(4):223–231. doi: 10.7150/ijms.4.223

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© Ivyspring International Publisher. This is an open-access article distributed under the terms of the Creative Commons License (http://creativecommons.org/licenses/by-nc-nd/3.0/). Reproduction is permitted for personal, noncommercial use, provided that the article is in whole, unmodified, and properly cited.

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Fig 6 — Down-regulation of hCG in Chlamydia trachomatis infected trophoblast. The effect of Chlamydia trachomatis on trophoblast hCG production during infection is depicted as hCG protein expression (A and B). Panel A shows reduction in β-hCG in infected trophoblast compared with uninfected cells. Less change was recorded in α-hCG. There was a significant decline (p < 0.05 *) in β-hCG production (B) in infected trophoblast (-x-) compared with uninfected (-■-) while the α-hCG showed no significant difference when Chlamydia trachomatis infected trophoblast (-▲-) is compared with uninfected trophoblast (-♦-). All values represent means ± SEM (n = 3).

Fig 6 — Down-regulation of hCG in Chlamydia trachomatis infected trophoblast. The effect of Chlamydia trachomatis on trophoblast hCG production during infection is depicted as hCG protein expression (A and B). Panel A shows reduction in β-hCG in infected trophoblast compared with uninfected cells. Less change was recorded in α-hCG. There was a significant decline (p < 0.05 *) in β-hCG production (B) in infected trophoblast (-x-) compared with uninfected (-■-) while the α-hCG showed no significant difference when Chlamydia trachomatis infected trophoblast (-▲-) is compared with uninfected trophoblast (-♦-). All values represent means ± SEM (n = 3).