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. Author manuscript; available in PMC: 2007 Oct 10.

Published in final edited form as: Virology. 2006 Nov 28;360(2):305–321. doi: 10.1016/j.virol.2006.10.028

FIG. 2 — Change of IRF-3 localization in the presence of ICP0. HEC-1-B cells were infected with SeV in the presence or absence of d106. (A) The cells were fixed at 2.5 h post infection and stained with mouse anti-ICP0 and rabbit anti-IRF-3 antibodies with appropriate secondary antibodies. (B) The cells were stained with mouse anti-p300 and rabbit anti-IRF-3 antibodies with appropriate secondary antibodies. (C) The cells were stained with mouse anti-CBP and rabbit anti-IRF-3 antibodies with appropriate secondary antibodies.

FIG. 2 — Change of IRF-3 localization in the presence of ICP0. HEC-1-B cells were infected with SeV in the presence or absence of d106. (A) The cells were fixed at 2.5 h post infection and stained with mouse anti-ICP0 and rabbit anti-IRF-3 antibodies with appropriate secondary antibodies. (B) The cells were stained with mouse anti-p300 and rabbit anti-IRF-3 antibodies with appropriate secondary antibodies. (C) The cells were stained with mouse anti-CBP and rabbit anti-IRF-3 antibodies with appropriate secondary antibodies.