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. 2006 Dec 1;4(3):321–325. doi: 10.1093/ecam/nel087

Table 2.

Effects of HSEt on changes in the levels of TBARS, HP, SOD, CAT, GPX and GSH in brain of normal and experimental rats (mean ± SD, n = 6)

Group SOD (UA) CAT (UB) GPx (UC) GSH (mg per 100 g tissue) TBARS (mM per 100 g tissue) HP (mM per 100 g tissue)
Control 7.75 ± 0.39a 3.21 ± 0.23a 3.43 ± 0.20a 36.01 ± 2.69a 1.09 ± 0.07 a 113.17 ± 4.39a
Control + HSEt (250 mg kg−1) 7.01 ± 0.27a 3.30 ± 0.29a 3.81 ± 0.18a 37.52 ± 2.72a 0.91 ± 0.09 a 109.01 ± 5.10a
NH4Cl treated (100 mg kg−1) 5.11 ± 0.43b 0.79 ± 0.06b 1.15 ± 0.07b 15.27 ± 1.43b 2.01 ± 0.14 b 132.70 ± 2.54b
NH4Cl + HSEt 7.32 ± 0.46c 2.74 ± 0.19c 2.61 ± 0.14c 27.01 ± 2.32c 1.21 ± 0.06c 118.42 ± 2.75c

ANOVA followed by DMRT. Values not sharing a common superscript (a, b, c) differ significantly at P ≤ 0.05.

Duncan procedure: ranges for the levels are 2.91, 3.06, 3.16, 3.22.

AAmount of enzyme required to inhibit 50% of NBT reduction per mg protein.

BMicromoles of H2O2 consumed per min per mg protein.

CMicromoles of GSH utilized per gram protein.