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(A) Tumor cell survival mechanism by Akt-mediated BAD phosphorylation followed by 14-3-3 chaperon-mediated pBAD degradation. (B) Tumor cell death mechanism in the presence of sense-p16 and antisense-uPAR bicistronic construct via inhibition of pAkt-mediated BAD phosphorylation followed by activation of caspase-mediated cell death.
