Table 2.
Source | Generation | Toxin | 10 mg/liter
|
100 mg/liter
|
||
---|---|---|---|---|---|---|
n | Mortality,* % | n | Mortality,* % | |||
QL4 | F1† | Cry1Ab | 21 | 62 | 17 | 65 |
Cry1Ac | 19 | 63 | 19 | 37 | ||
Others‡ | F1 | Cry1Ab | 100 | 100 | 96 | 100 |
Cry1Ac | 99 | 100 | 96 | 100 | ||
QL4 | F2§ | Cry1Ab | 100 | 4 | 100 | 0 |
Cry1Ac | 100 | 0 | 100 | 4 | ||
QL4 | F4 | Cry1Aa | 100 | 0 | 96 | 0 |
QL4 | F8 | Cry1Ab | 40 | 0 | 40 | 0 |
Cry1Ac | 40 | 0 | 41 | 0 | ||
QL4 | F10 | Cry1F¶ | 50 | 2 | 50 | 0 |
Mortality after 5 days was adjusted for control mortality.
F1 progeny from family QL4 were produced by one NO-QA female and one LAB-P male; pooled mortality of F1 (56%) was not significantly different from 50% (χ2 = 1.32, df = 1, P > 0.2).
Five other F1 families from single-pair crosses of NO-QA × LAB-P were tested simultaneously with QL4.
Thirty-one F1 survivors of exposure to Cry1Ab or CryAc were pooled and mated among themselves to produce F2.
Concentrations for Cry1F were 1 and 10 ml of formulated Cry1F per liter.