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. Author manuscript; available in PMC: 2007 Oct 2.
Published in final edited form as: Nature. 2007 Feb 1;445(7127):554–558. doi: 10.1038/nature05503

Figure1. Mutations in lsn and vps36 disrupt the anterior localisation of GFP-Stau and bcd mRNA.

Figure1

a and b GFP-Staufen (green) localisation to the anterior cortex of wildtype stage 11 (a) and stage 13 (b) oocytes. The actin cortex is labelled with Rhodamine-Phalloidin (red).

c bcd mRNA localisation in a freshly-laid wildtype egg.

d and e Homozygous vps22/lsn germline clones, showing the absence of GFP-Stau at the anterior pole of the oocyte at stage 10B (d) and at stage 13 (e).

f bcd mRNA localisation in an embryo derived from a vps22/lsn germline clone.

g and h Homozygous vps25Pb2931 germline clones, showing the absence of GFP-Stau at the anterior pole of the oocyte at stage 10B (g) and at stage 13 (h).

i and j Homozygous vps36 (l(3)L5212) germline clones, showing the absence of GFP-Stau at the anterior pole of the oocyte at stage 11 (i) and at stage 13 (j).