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PE-induced vasoconstriction in the cavernous artery in the absence or presence of 1, 10 or 100 nM prazosin (non-selective antagonist, n=7–8), RS 100329 (α1A-AR antagonist, n=5–6), Rec 15/2615 (α1B-AR antagonist, n=3–4) or BMY 7378 (α1D-AR antagonist, n=6). All PE response data were taken from second CRCs in the absence or presence of antagonist.
