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. 2006 Oct 3;149(6):657–665. doi: 10.1038/sj.bjp.0706897

Figure 4.

Figure 4

(a) Normalized traces comparing INa from mutant and wild-type VGSCs expressed in tsA201 cells. (b) Representative traces of INa through mutant (R1623Q) VGSCs in the presence of 0 or 50 μM resveratrol. (c) Representative traces of INa through R1623Q VGSCs in the presence of 0 or 10 μM quercetin. (d) Peak and late INa through R1623Q VGSCs in the presence of 50 μM resveratrol or 10 μM quercetin. All groups were normalized to their controls. Late R1623Q INa was blocked to a greater extent than peak INa during application of 50 μM resveratrol (n=7) and late R1623Q INa was blocked to the same extent as peak INa during application of 10 μM quercetin (n=10). *P<0.001.