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. 2007 Nov;18(11):4387–4396. doi: 10.1091/mbc.E07-05-0486

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© 2007 by The American Society for Cell Biology

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Figure 4. — UNC-11 (AP180) is required for GLR-1 accumulation in lin-10 mutants but not in rab-10 mutants. GLR-1::GFP fluorescence was examined in unc-11 mutants (A), lin-10 unc-11 double mutants (B), and rab-10 unc-11 double mutants (C). The mean size (D and E) of fluorescent clusters is plotted for adults of the given genotype. (F) The mean spontaneous reversal frequency (number of reversals per minute over a 5-min period) is plotted for the given genotype. Mutations in unc-11 suppress the accumulation of GLR-1 in lin-10 mutants, and they increase the accumulation of GLR-1 in rab-10 mutants. They also completely suppress the spontaneous reversal defect in lin-10 mutants. ***p < 0.001, **p < 0.01 by ANOVA followed by a Bonferroni multiple comparison test for the indicated comparisons. Bar, 5 μm. n = 15–20 animals for each genotype.