FIG. 2.

Complementation of JG736. The observed phenotype was linked to the disruption of lpp by complementing the JG736 colonization defect. The parent strain, HM21R, and JG736, each carrying either the broad-host-range vector pMMB207 or pAS11 (pMMB207 containing the complementing region) were tested against the competitor strain, HM21RS, and assessed at 72 h. The CI [calculated as (mutant_output/competitor_output)/(mutant_input/competitor_input)] for each animal is shown. Horizontal lines represent median CI values. Asterisks indicate a statistically significant difference between data sets (P < 0.05 by the Mann-Whitney test); ns, not significantly different.