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. 2007 Jul 25;81(19):10792–10803. doi: 10.1128/JVI.00196-07

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Copyright © 2007, American Society for Microbiology

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FIG. 6. — Alterations in modifications of emerin in cells infected with HSV-1 mutants. Shown are digital images of Western blots of lamina fractions of Vero (A) or HEp-2 (B) cells that had been infected at 5 PFU/cell for 16 h with wild-type HSV-1(F) (panel A, lanes 2 and 7; panel B, lane 2), US3-null virus vRR1202 (panel A, lane 3, and panel B, lane 3), homologous repair of vRR1202 (panel A, lane 4; panel B, lane 4), recombinant virus expressing catalytically inactive US3 vRR1204 (panel A, lane 5; panel B, lane 5), UL34-null virus vRR1072(tk+) (panel A, lane 8; panel B, lane 6), and homologous repair of vRR1072(tk+) (panel A, lane 9; panel B, lane 7). The blots were probed with monoclonal antibody to emerin.