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. 2006 Jul 17;174(2):267–275. doi: 10.1083/jcb.200510156

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Copyright © 2006, The Rockefeller University Press

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Figure 4. — Cdc42 Rho GTPase and p38 MAPK signaling pathways are involved in α-herpesvirus–induced varicosity formation. Percentage of axons with varicosities in mock-treated two-chamber models or at 16 h after incubation with an amount of UV-inactivated PRV particles equivalent to 2 × 10⁶ PFUs of WT-PRV, in the presence of inhibitors directed against (A) small Rho GTPases (C. difficile toxin B), Rho (Y27632), Rac1 (Rac1 inhibitor), or Cdc42 (secramine A) or (B) ERK (U0126), JNK (JNK inhibitor II), or p38 MAPK signaling (SB203580). Data shown represent means ± SEM of triplicate assays. Percentages indicated by the same letter do not significantly differ (α = 0.05).