Table 4.

Description of cases of “true positive” findings (n = 12) out of 217 patients operated for spinal deformity

	Region	Pathology	Surgery	Duration hours	IOM modality	Baseline exam	Changes of MIOM	Neurological deterioration	Duration	Recovery
True positive cases
R.A., f, 54y	T8 − L3	Scoliosis postraumatic	Decompression, correction and fusion, dvd	12.6	csT10EP cmTA,AHEP sT10mTA,AHEP nPNsT10EP	All available, but pathologic	After distraction T12 − L1 loss of all potentials of cmEP	Deterioration of existing partialConus syndrome	?	?
H.S., f, 75y	L3 − S1	Scoliosis degenerative	Decompression and fusion, d	3.0	cmVM,TA,AHEP nPNcEP cont EMGVM,TA,AH	cmVM,TAEP right not available	Reduction of amplitude of cmEP VM right during decompression	Partial sensomotor deficits L4 right	Hours	Complete
J.K., f, 44y	T5 − L2	Scoliosis after tumour resection	Columnotomy correction and fusion, d	7.25	csT7,L1EP cmVM,TAEP sL1mVM,TAEP sL1sT7EP nTNsT7EP nTNcEP cont EMGVM,TA	All available	At the end of columnotomy loss of cmEP, reduction of D-wave. Continuous recovery of D-wave, but pathologic cmEP at the end of operation	Partial paraparesis	Hours	Complete
S.K., f, 74y	T7 − L3	Scoliosis neuromuscular	Decompression, osteotomie T12, correction and fusion, d	9.5	csL1EP cmVM,TAEP nFN,PNsL1EP nFN,PNcEP cont. EMGVM,TA	All available, except not reproducible ncEP	Alteration of cmEP of VM left during ped. screwing	Mild partial sensomotor deficits L3 left	Hours	Complete
F.C., m, 57y	T9 − L2	Scoliosis posttraumatic	Correction and fusion, dvd	10.0	csT8,L1EP cmVM,TA,AH,BREP smVM,TA,AH,BREP sT8sL1EP nTN,MNsL1,T8EP nTN,MNcEP cont. EMGVM,TA,AH,BR	Normal cmEP, pathologic ncEP	Fading away of all cmEP	Cortical event (disturbance of conciousness)	Hours	Complete
R.G., w, 80y	T5 − L5	Scoliosis degenerative	Decompression, correction and fusion	5.5	cmVM,otherEP nFNcEP cont. EMGVM,other	All available, but pathologic	After correction massive reduction of amplitude of cmEP VM, partial recovery	Partial sensomotor deficits L3 + L4	Hours	Complete
N.G., m, 8y	L4 − S2	Scoliosis congenital anomaly	Correction and fusion, d	8.0	cmVM,TA,AHEP nTNcEP cont. EMGVM,TA,AH Ped. screw stimulation	Pathological baseline for all modalities	Reduction of amplitude of cmEP during correction	Partial paraparesis	6 weeks	Complete
W.C., f, 14y	T3 − L2	Scoliosis idiopathic	Correction and fusion, d	3.75	csT1,T12EP cmTA,AHEP sT1mTA,AHEP nTNcEP nTNsT12EP cont. EMGTA,AH	All available	Associated with bleeding partial loss of motor and sensory EP, no recovery	Partial paraparesis	2 h	Complete
Y.F., m, 16y	T2 − L2	Scoliosis congenital anomaly	Correction and fusion, d	5.0	csT10,C5EP cmTA,AHEP smTA,AHEP sT0sC5EP nTNsC5EP nTNcEP cont. EMGTA,AH	All available, but pathologic	Loss of all EP associated to massive blood loss, only partial recovery of EP	Partial paraparesis	Hours	Complete
P.L., f, 13y	T3 − L2	Scoliosis	Correction and fusion, d	4.25	csT12,T8EP cmTA,AHEP sT12mTA,AHEP nTNsT12,8EP nTNcEP cont. EMGTA,AH	All available	Partial loss of all EP, no recovery.	Partial paraparesis	Hours	Complete
M.R., m, 16y	L3 − L5	Scoliosis neuromusculararthrogryposis	Correction and fusion, d	2.6	cmVM,TAEP nPNcEP cont. EMGVM, TA	Normal cmEP, pathologic ncEP	Loss of all EP.	Cardiopulmonary decompensation due to intravasal coagulopathy	Exitus letalis
V.L., m, 73y	C2 − T8	Scoliosis neuromuscular	Osteotomie C7/T1, correction and fusion, d	7.0	cmTA,D,TM,ADMEP nMN,TNcEP cont. EMGTA,D,TM,ADM	All available, but pathologic	Massive alteration of cmEP TM right associated to correction, no recovery	Partial sensomotor deficit C7 right	3 months	Complete

The onset of new neurological deficit could be predicted based upon IOM findings