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. 2006 Jun 20;77(10):1169–1176. doi: 10.1136/jnnp.2006.093443

Table 1 Nerve conduction findings at the last electrophysiological examination (in 1983 for patient II‐13 and in 2005 for the remaining patients).

Patient Motor Sensory
Nerve DML (ms) MCV (m/s) CMAP (mV) SCV (m/s) SNAP (μV)
II‐13 Left brachial plexus 15.1* NA 1*
Left radial 8.7 NA 2
Left median A A A A A
Left peroneal A A A
III‐24 Right median 9.9 17.6 0.1 A A
Right radial A A
Right peroneal A A A
10.1† NA 0.1†
Left femoral 11.6 NA 0.1
Right femoral 8.4 NA 0.1
IV‐17 Right median 10.1 20.7 3.1 A A
Right ulnar 4.6 19.2 6.3 27.8 0.9
Right peroneal 9.1 16.9 1.1
Right tibial 12.2 15.8 2.3
Right sural A A
IV‐18 Right median 8.8 22.7 7.8 A A
Right ulnar 7.8 20.3 4.8 19.9 0.3
Right peroneal 12.5 17.5 0.3
Right tibial 10.2 20.4 0.1
Right sural A A

A, absent; CMAP, compound muscle action potential; DML, distal motor latency; MCV, motor conduction velocity; NA, not applicable; SCV, sensory conduction velocity; SNAP, sensory nerve action potential.

*To biceps muscles.

†To tibialis anterior muscle.

Patient III‐24 underwent three previous electrophysiology examinations in 1977, 1990 and 1999. The recordings were destroyed when one of our hospital buildings collapsed in 1999. According to clinical record data, we can merely state that the peroneal nerve has been inexcitable at the ankle since 1977, and that the slowing of MCVs of the median nerve has remained stable.