Skip to main content
. Author manuscript; available in PMC: 2008 Sep 21.
Published in final edited form as: Neuroscience. 2007 Jul 17;148(4):978–995. doi: 10.1016/j.neuroscience.2007.07.013

Fig. 9.

Fig. 9

Changes in discharge rate (A and B- mean values for 0.5 s±standard errors and C – original examples) induced by iontophoretic application of glutamate (20 nA) in rats during urethane anesthesia and in awake conditions with DA receptor blockade. n is the number of cells (=applications). The effect of time was significant and similar for both conditions (F24,574=5.44 and F22,528=4.55, p<0.001), but its amplitude was significantly larger in awake than anesthetized conditions (see text). As shown in C, GLU in each neuron induced excitation, which developed with 1–2 s latencies. This excitation manifested as increased discharge rate, often accompanied by increased bursting.