Skip to main content
. Author manuscript; available in PMC: 2008 Dec 1.
Published in final edited form as: Neurobiol Aging. 2007 Aug 27;28(12):1840–1852. doi: 10.1016/j.neurobiolaging.2007.07.012

Figure 9.

Figure 9

Brain proteins from Atrn null mutants show elevated oxidative damage. Oxyblot assay of brain lysates from 3-month old mice revealed increased levels of protein carbonyls in Atrnmg-3J/mg-3J mutants compared to Atrnmg-3J/+ siblings. Blots were stripped and blotted for GAPDH as loading control. Signal intensity was quantified using ImageJ software and plotted as average ratio of the Oxyblot signal/GAPDH signal by genotype (B) to quantify differences in oxidatively damaged proteins. Atrnmg-3J/mg-3J ratio was normalized against Atrnmg-3J/+.