Table II.
Genetic Analyses of Phototactic Mutant Strains
| Isolation name | Allele designation | Ptx−/Mot− segregation* | Ptx−/Arg+ segregation‡ | Map position | ||||
|---|---|---|---|---|---|---|---|---|
| 7-1 | mia1-1 | 136 | − | L.G. II (24:0:0 to act1) | ||||
| 13-5 | mia2-1 | 119 | − | L.G. XVIII (31:0:6 to spr1) | ||||
| AT1-2 | pf9-4 | 56 | 21 | L.G. XII/XIII (32:0:0 to pf9) | ||||
| AT1-25 | ptm1-1 | 70 | 29 | L.G. II (73:0:8 to fsr1) | ||||
| AT2-121 | ptm2-1 | 52 | 21 | Unknown§ | ||||
| 8-10 | ptm3-1 | 102 | − | L.G. XVIII (61:2:14 to spr1) | ||||
| 8-14 | ptm3-2 | 43 | − | L.G. XVIII‖ | ||||
| 14-9 | ptm3-3 | 25 | − | L.G. XVIII‖ |
Number of tetrads in the phototaxis (Ptx−) and motility (Mot−) phenotypes segregated together and as 2+:2−.
Number of tetrads in which the phototaxis (Ptx−) and arginine prototrophy (Arg+) phenotypes segregated together and as 2+:2−.
This mutation did not show linkage to other genetic markers, but was centromere linked based on mapping to ac17 (8:7:0) and pyr1 (10:5:0).
These mutations were not directly mapped because complementation and recombination analyses described in the text showed that they were allelic with the ptm3-1 mutation.