Skip to main content
. 1998 Oct 5;143(1):171–181. doi: 10.1083/jcb.143.1.171

Figure 3.

Figure 3

Absence of NF-H does not markedly affect growth in motor axon caliber. (A) Cross sections of L5 motor (ventral root) and sensory (dorsal root) axons from wild type (+/+), NF-H heterozygous (+/−), and NF-H homozygous (−/−) mutant mice. Sections are from 4- and 9-wk-old mice as indicated. (B and C) Absence of NF-H yields a partial loss of motor and sensory axons in early postnatal life. Counts of small (<4 μm diameter; black bars) and large (≥4 μm diameter; crosshatched bars) axons in L5 (B) motor and (C) sensory root axons from 9-wk-old mice with zero, one, or two disrupted NF-H alleles. Counts are averages from three to four animals for each genotype and age. Bar, 10 μm.