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. 1997 Nov 3;139(3):735–747. doi: 10.1083/jcb.139.3.735

Table IV.

Summary of the Involvement of the Various RARs and RXRs in the Transduction of the Retinoid Signal in F9 Cells, as Deduced from the Present and Previous Studies of RAR and RXR Mutant Cells and the Use of Receptor-specific Retinoids

Retinoid-induced events and RXR/RAR pairs capable of transducing the signal Role of RXRs Role of RARs
Primitive endodermal differentiation RARγ
 RARγ ligand active on its own at saturating
RXR ligand inactive on its own (Roy et al., 1995)    concentration (Taneja et al., 1996)
 RARγ−/− cells differentiate very poorly   (Boylan et al., 1993)
RXRα/RARγ in all instances RXRα−/− cells differentiate very poorly  (Clifford et al., 1996)  synergizes with RXRα at suboptimal ligand   concentration  (Clifford et al., 1996)
 weakly hindered by RARα in WT cells   (Taneja et al., 1996)
RXR ligand is required at suboptimal concentration RARα
 of RARγ ligand (Roy et al., 1995; Taneja et al., 1996)  RARα ligand inactive on its own (Taneja et al., 1996)
 RARα−/− cells differentiate normally   (Boylan et al., 1995)
 can poorly replace RARγ provided RXRα is activated
RXRα can be poorly replaced by RXR(β+γ) RARβ
 provided that RARγ is present  inactive or very poorly active (Taneja et al., 1996)
Visceral endodermal differentiation RARγ
RXR ligand inactive on its own RXR ligand is required at suboptimal concentration  of a RAR ligand RXRα can be efficiently replaced by RXR(β+γ)  provided that RARγ is present RXRα prevents efficient synergism between RXR(β + γ) and RARγ  indispensable in WT cells
 RARγ ligand active on its own at saturating   concentration
RXRα/RARγ in WT cells  synergizes with RXRs at suboptimal ligand   concentration
 hindered by RARα in WT cells
RARα
RXR(β+γ)/RARγ in absence  RARα ligand inactive on its own or very poorly active
 of RXRα (efficiently)  RARα−/− cells differentiate normally
 synergizes with RXRα in the absence of RARγ
RXRα/RARα or RARβ in  hindered by RARγ in WT cells
  absence of RARγ (efficiently) RARβ
 RARβ ligand inactive on its own or very poorly active
 synergizes with RXRα in the absence of RARγ
 blocked by RARγ in WT cells
Inhibition of proliferation RARγ
 not indispensable in WT cells
RXRα/RARγ or RARα in WT cells  RARγ ligand active on its own at saturating   concentration
RXR ligand inactive on its own  synergizes with RXRs at suboptimal ligand   concentration
 partially hindered by RARα in WT cells
RXRα/RARβ in absence of RARγ RXR ligand is required at limiting concentration  of RAR  ligand RARα
 RARα ligand inactive on its own
 synergizes with RXRα in WT cells
RXR(β+γ)/RARγ in absence  of RXRα RXRα can be partially replaced by RXR(β+γ)  provided that RARγ is present  partially hindered by RARγ in WT cellsRARβ
 RARβ ligand inactive on its own
 synergizes with RXRα in the absence of RARγ
 blocked by RARγ in WT cells
Induction of proliferation in RARα
RXRα−/−/RARγ−/−cells  RARα ligand active on its own
RXR ligand inactive on its own RARβ
RXR(β+γ)/RARα  RARβ ligand active on its own
RXR(β+γ)/RARβ  synergizes with RXR(β+γ)
Apoptosis RARγ
 indispensable in WT cells
RXRα/RARγ in WT cells RXR ligand inactive on its own  RARα ligand active on its own at saturating ligand   concentration
RXR(β+γ)/RARγ in absence of  RXRα, but very inefficiently RXRα ligand is required at suboptimal ligand  concentration of RAR ligand  synergizes with RXRα at suboptimal ligand   concentration
 hindered by RARα in WT cells
RARα
RXRα/RARα or RARβ in absence of  RARγ,  but inefficiently RXRα can be poorly replaced by RXR(β+γ)  provided that RARγ is present  RARα ligand inactive on its own  weakly synergizes with RXRα in the absence of RARγ  blocked by RARγ in WT cells
RARβ
 RARβ ligand inactive on its own
 weakly synergizes with RXRα in the absence of RARγ
 blocked by RARγ in WT cells