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Dose response of RIP14 activation by retinoids. (A) JEG-3 cells were transfected as in Fig. 1 with RIP14, RXR, and hsp27–EcREx5 Δ mouse mammary tumor virus–luciferase and treated with 0–10 μM TTNPB ± 1 μM 9-cis-RA and 0–10 μM tRA ± 1 μM 9-cis-RA. (B) JEG-3 cells were transfected with CDM8 as a carrier plasmid and the β2RARE x3 TK luc reporter plasmid, and the response of endogenous RAR to either 0–1 μM TTNPB or tRA was determined.
