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Schematic representation of a possible mechanism of action of JH on adult neuroblast proliferation. As for vertebrate hormones, JH could act on the protooncogenes c-myc and max (39–41) and induce the activation of ODC gene (42, 43). The resulting putrescine production could regulate protooncogene expression (47–50) and/or act on cyclins involved in cell cycle control (51, 52).
