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. 2007 Oct 3;81(24):13694–13699. doi: 10.1128/JVI.01646-07

FIG. 3.

FIG. 3.

The CDAs present in A3G are critical for inhibition of RSV infectivity. (A) This infection experiment was performed as described in the legend for Fig. 1A, except that the indicated wild-type or mutant forms of human A3G were analyzed. The pK/β-arr plasmid, which expresses HA epitope-tagged β-arr, was again used as a control (lane 1). (B) Western analysis of the intracellular expression levels of the β-arr and A3G protein variants described for panel A. (C) Western analysis of the intravirion expression levels of β-arr and the A3G protein variants described for panel A. This Western blotting, which was performed as described in the legend for Fig. 2, analyzed RSV virions released from the same QCl-3 cells described for panel B. (D) Western analysis of the levels of RSV capsid protein expression in the pelleted virions analyzed as described for panel C. The results of a representative experiment are shown.